Zombie Apocalypse Show isn't Real Enough (The Walking Dead on AMC)

Maggie is on foot but the Tyrese carrying the baby thing is still a theory

 

Well, I thought that too until the previews where it shows Tyrese in a world of shit and no baby around.

In the previews, Maggie looked like she just stepped off the crazy bus...

 

well maybe the previews are just showing that the baby isn't dead..... yet.

 

I think Maggie finds the bus overturned , that's what I got out of that

 

Beth is 18 years old on the show. The actress who plays Beth (Emily Kinney) is 28 years old.

 

I hope Darryl gets some! So far I don't think he has tapped anything but zombies!

 

X2

Really!? Would've never imagined her being that much older than me

 

He probably tapped Carol - who can be mistaken for a walker at times.

 

Good point! lol

 

That was intense....

Michonne first pet zombies were her BF and the BF's friend?

Glenn seem to be in the up in the tower..

Just crazy mayhem, the preview didnt show much. Can't wait on the next episode.

 

another lesson learn

 

ed

 

HAHAHAHA that's what that little turd gets

 

I think I'm right when I say that the baby gets killed in the graphic novel. Of course, keeping her alive wouldn't be the first thing they've done that's strayed away from the original story line.

 

My theory on the baby. The baby is not only alive but is not infected. A baby born after the outbreak would have been protected in the womb. A vaccine will be made using her blood. She must be kept alive at all costs. She survived a walker bite and now everyone knows she is not infected. Or she is dead and they just didn't show it.
Placental mechanisms[edit]


The placenta functions as an immunological barrier between the mother and the fetus.


The placenta functions as an immunological barrier between the mother and the fetus, creating an immunologically privileged site. For this purpose, it uses several mechanisms:

• It secretes Neurokinin B containing phosphocholine molecules. This is the same mechanism used by parasitic nematodes to avoid detection by the immune system of their host.[2]
• Also, there is presence of small lymphocytic suppressor cells in the fetus that inhibit maternal cytotoxic T cells by inhibiting the response to interleukin 2.[1]
• The placental trophoblast cells do not express the classical MHC class I isotypes HLA-A and HLA-B, unlike most other cells in the body, and this absence is assumed to prevent destruction by maternal cytotoxic T cells, which otherwise would recognize the fetal HLA-A and HLA-B molecules as foreign. On the other hand, they do express the atypical MHC class I isotypes HLA-E and HLA-G, which is assumed to prevent destruction by maternal NK cells, which otherwise destroy cells that do not express any MHC class I.[3] However, trophoblast cells do express the rather typical HLA-C.[3]
• It forms a syncytium without any extracellular spaces between cells in order to limit the exchange of migratory immune cells between the developing embryo and the body of the mother (something an epithelium will not do sufficiently, as certain blood cells are specialized to be able to insert themselves between adjacent epithelial cells). The fusion of the cells is apparently caused by viral fusion proteins from endosymbiotic endogenous retrovirus (ERV).[4] An immunoevasive action was the initial normal behavior of the viral protein, in order to avail for the virus to spread to other cells by simply merging them with the infected one. It is believed that the ancestors of modern viviparous mammals evolved after an infection by this virus, enabling the fetus to better resist the immune system of the mother.[5]

Still, the placenta does allow maternal IgG antibodies to pass to the fetus to protect it against infections. However, these antibodies do not target fetal cells, unless any fetal material has escaped across the placenta where it can come in contact with maternal B cells and make those B cells start to produce antibodies against fetal targets. The mother does produce antibodies against foreign ABO blood types, where the fetal blood cells are possible targets, but these preformed antibodies are usually of the IgM type,[6] and therefore usually do not cross the placenta. Still, rarely, ABO incompatibility can give rise to IgG antibodies that cross the placenta, and are caused by sentitization of mothers (usually of blood type 0) to antigens in foods or bacteria.[7]
Other mechanisms[edit]

Still, the placental barrier is not the sole means to evade the immune system, as foreign fetal cells also persist in the maternal circulation, on the other side of the placental barrier.[8]
The placenta does not block maternal IgG antibodies, which thereby may pass through the human placenta, providing immune protection to the fetus against infectious diseases.
One model for the induction of tolerance during the very early stages of pregnancy is the Eutherian Fetoembryonic Defense System (eu-FEDS) hypothesis.[9] The basic premise of the eu-FEDS hypothesis is that both soluble and cell surface associated glycoproteins, present in the reproductive system and expressed on gametes, suppress any potential immune responses, and inhibit rejection of the fetus.[9] The eu-FEDS model further suggests that specific carbohydrate sequences (oligosaccharides) are covalently linked to these immunosuppressive glycoproteins and act as “functional groups” that suppress the immune response. The major uterine and fetal glycoproteins that are associated with the eu-FEDS model in the human include alpha-fetoprotein, CA125, and glycodelin-A (also known as placental protein 14 (PP14)).
Regulatory T cells also likely play a role.[10]
Also, a shift from cell-mediated immunity toward humoral immunity is believed to occur.[11] womb.

 

Jesus fucking Christ....... You must be bored as hell lol.

 

they can't kill the baby, think of all the marketing on items like this they will miss out on!

 

They already did

 

Parents should be charged with abuse if they put that on their baby.

But dang what happened to the evolution thread??